Neutrophil Extracellular Traps Participate in Cardiovascular Diseases: Recent Experimental and Clinical Insights
Neutrophils as Regulators of Cardiovascular Inflammation
Recruitment of classical monocytes can be inhibited by disturbing heteromers of neutrophil HNP1 and platelet CCL5
Chrono-pharmacological Targeting of the CCL2-CCR2 Axis Ameliorates Atherosclerosisz
Double-Strand DNA Sensing Aim2 Inflammasome Regulates Atherosclerotic Plaque Vulnerability
Cathepsin G Controls Arterial But Not Venular Myeloid Cell Recruitment
Neutrophils as protagonists and targets in chronic inflammation
Basic research: Standardizing animal atherosclerosis studies to improve reproducibility
